CONTENTS.
xxv
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degeneration of muscles.—Fatty infiltration. Intestines; structure and functions of the villi. Reabsorption and retention of the chyle. Liver; intermediate interchange of matter by means of the biliary ducts. Fatty liver.—Fatty metamorphosis. Glands; secretion of sebaceous matter and milk (colostrum). Granule-cells and granule-globules. Inflammatory globules. Arteries; fatty usure and atheroma in them. Fatty débris. || | |
| LECTURE XVI.—A more Precise Account of Fatty Metamorphosis. | 383 |
| Fatty degeneration of muscles. Fatty metamorphosis of the substance of the heart. Formation of fat in the muscles in disiortions.—Corpus luteum of the ovary. Fatty metamorphosis of pulmonary epithelium. Yellow softening of the brain. Arcus senilis.—Optical properties of fattily degenerated tissues. Renal epithelium in Bright's disease. Successive stages (cloudy swelling, fatty metamorphosis, fatty detritus (débris), atrophy). Inflammatory globules. Similarity of the result in inflammatory and non-inflammatory changes.—Atheromatous process in arteries. Its relation to ossification. Inflammatory character of the process; its analogy with endocarditis. Formation of the atheromatous deposit. Appearance of cholestearine. Arterio-sclerosis. Endoarteritis. Calcification and ossification of arteries.—Mixed, activo-passive processes. | |
| LECTURE XVII.—Amyloid Degeneration. Inflammation. | 409 |
| Amyloid (lardaceous or waxy) degeneration. Different nature of amyloid substances: concentric and laminated amyloid bodies (brain, prostate), and amyloid degeneration properly so called. Its course. Commencement of the affection in the minute arteries. Waxy liver. Cartilage. Dyscrasic (constitutional) character of the disease. Intestines. Kidneys: the three forms of Bright's disease (amyloid degeneration, parenchymatous, and interstitial nephritis), Lymphatic glands. Functional disturbances of the affected organs.—Inflammation. The four cardinal symptoms and their predominance in the different schools: the thermic and vascular theory; the neuro-pathologists, exudatious. Inflammatory stimuli. Lesion of function. Exudation as a consequence of the activity of the tissues; mucus and fibrine. Inflammation as a complex irritative process. Parenchymatous and exudative (secretory) form. | |
| LECTURE XVIII.—Normal and Pathological New Formation. | 438 |
| The theory of continuous development in opposition to the blastema and exudation theory—Connective tissue and its equivalents as the most general germ-store of new formations. Correspondence between embryonic and pathological new formation. Cell-division as the most general starting-point of new-formations.—Endogenous formation. Physalides. Brood-cavities.—Different tendencies of new-formations. Hyperplasia, direct and indirect. Heteroplasia. Pathological formative cells. Difference in their size and in the time required for their full development.—Description of the development of bone as a model formation. Differ- |
